User:Saltrabook/The OM Patient Record

The occupational history
An inadequate occupational history may result in labelling an illness as not "occupational" when it is, or vice-versa. It may result in missing the diagnosis altogether and in losing an opportunity (or even the last or only opportunity) of making the patient better and improving the fate of other workers. As in other aspects of medical history taking, two important lines of enquiry that must be followed are questions designed to elicit information about the:
 * 1)  symptoms of existing underlying pathology
 * 2)  background which may increase the likelihood of such pathology

Taking an Occupational History ppt

Development of the disease
In relation to the symptoms themselves, questioning may elicit : But, most importantly in taking occupational history it is important to determine exposure :
 * The organ system and type of pathological process involved e.g. wheeze, chest tightness and cough would suggest asthma, red itchy spots on a dry skin would suggest eczema/dermatitis.

What does an 'electro-plater' do, or a 'ram-block packer'!? The full job description may need to go back several years - mesothelioma for example can occur 40 years after first exposure to asbestos. Indeed in some circumstances it is important to take an occupational history going as far back as the patient’s first job (an environmental exposure history has rarely had to extend even to the pre-school years - as in the case of children who had played on tips containing asbestos or other hazardous substances). Colloquial terms may be used for chemicals e.g. "trike" (trichloroethylene), "perk" (perchloroethylene), or "monkey dung" (brown asbestos). Confirmation of the identity of a chemical might involve asking the patient to bring in a label or a hazard data sheet. For example if the history is suggestive of exposure to formaldehyde, then the label of the drum should confirm this:
 * 1) A job title is not adequate, a full description of what the job entails is a bare minimum.
 * 1) The identity of any chemical exposures or other hazards must be clearly established.
 * 1)  It is important to try and determine the duration and intensity of exposure. Determining when exposure started, when it finished and how often it was repeated may be tedious but not very difficult. However determining the intensity of exposure by history taking is very difficult but the following are useful guides :

Specific questions on the job content

 * 1) How was the task done e.g. was an adhesive applied with a paint brush while the patient leant over it?
 * 2) Was the dust concentration so bad that one could not see clearly through it, or the noise so loud that communication was difficult?
 * 3) If control of exposure to a chemical appeared poor, what sort of quantities of it were handled?
 * 4) Was there any attempt at segregating harmful tasks, or providing local exhaust ventilation? In the example shown alongside, there was significant exposure to a serious hazard, but without any adequate attempt at controlling exposure.
 * 5) Special questions are warranted in relation to personal protection. A response that the worker wears a mask is about as informative as a patient saying that he takes wee white pills.
 * 6) Many masks offer no protection whatsoever against gases vapours or dust e.g. surgical masks or old gauze masks, while "Nuisance Dust" masks of the sort often sold in DIY shops are not much better.
 * 7) Different categories of harmful agents tend to have different and specific forms of personal protection. For example, masks designed for respirable dust will not protect from sulphur dioxide or solvent exposure.

Use of preventive measures

 * 1) Gloves that may be suited to provide some protection against cement dust might not be of  any use at all if exposed to petrol. The patient in the image (right) had high quality thick industrial black rubber gloves - but unfortunately they offered no protection from the alkylating agent which she was handling.
 * 2) The control measures e.g. local exhaust ventilation and personal protective equipment need to be regularly maintained. Ask the worker whether they are and who is responsible for this. in the example (left), a wide range of high quality, well maintained protective measures are in use:

Processing the historical information, and further questions
There are a number of similarities between the processing of information in a clinical occupational medical history and the decisions regarding criteria for causal association that one encounters in epidemiology. Indeeed many good occupational physicians practise both clinical medicine and epidemiology within the specialty of occupational medicine. Analogies between determining causality in an epidemiologic context (the Bradford-Hill criteria) in population studies, and when dealing with an individual patient in a clinical context.

Epidemiologic criteria for causality (Clinical questions)

 * 1) Temporality
 * 2) When in relation to exposure do / did the symptoms start?
 * 3) Reversibility
 * 4) Do the symptoms improved when no longer exposed e.g. on holiday?
 * 5) Exposure-response
 * 6) Are the symptoms especially worse when undertaking tasks or in areas with high exposures?
 * 7) Strength of association
 * 8) Do other workers / patients suffer from similar symptoms associated with the same exposures?
 * 9) Specificity
 * 10) What other exposures / causal factors could be responsible for the same symptoms? (Smoking perhaps?)

Other data, or information processing

 * 1) Consistency
 * 2) Are there other reports of the same symptoms associated with or caused by the same exposure?
 * 3) Analogy
 * 4) Even if there is no evidence to hand of identical exposures or circumstances resulting in the same symptoms, have similar agents/ chemicals of similar structure been implicated in the same symptoms of  for example ... dermatitis, ... or asthma.
 * 5) Biological plausibility
 * 6) Do the symptoms 'add up' in terms of what is known about the mechanisms of disease?

Establishing the conclusion and further advice to the patient
A synthesis of the clinical symptoms´ development in parallel with the exposures on the workplace will be assessed together with the available scientific evidence of a possible causal relation between the disease and the workplace exposures. Then comes the question about notification, further prevention and advice to the patient about work/education in the future,